Commentary
See article on page 613HCV in hepatic failure: West and East do not meet
| The first 150 words of the full text of this article appear below. |
Despite the identification of hepatitis C virus (HCV) as
an aetiological agent of blood borne non-A, non-B (NANB) hepatitis a
decade ago, its role in fulminant NANB hepatitis is still
uncertain.1 In the West, HCV is only infrequently
associated with NANB fulminant hepatitis. Thus, in studies from the
USA, positive serum HCV RNA was reported in none of 15 cases by Wright
and colleagues2 and in only two (12%) of 17 cases by
Liang et al.3 Similarly, none
of the 23 and 30 NANB cases in the French and English series, respectively, was positive for HCV infection.4 5 In
contrast, the prevalence of serological markers of HCV infection in
NANB fulminant hepatitis is much higher in the Oriental countries. In
an early study from Japan, Muto et al found
that seven (58%) of 12 patients with NANB fulminant hepatitis were
anti-HCV (anti-C100-3) positive6; findings which were
later confirmed by Yangi et al who reported
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- Fulminant hepatic failure in acute hepatitis C: increased risk in chronic carriers of hepatitis B virus
- C M Chu, C T Yeh, and Y F Liaw
Gut 1999 45: 613-617.[Abstract] [Full Text] [PDF]
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