© 2004 by BMJ Publishing Group Ltd & British Society of Gastroenterology
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In this issue we have an important original article which we felt would be worth supporting with both a leading article focusing on the molecular biology as well as a commentary setting this in context for clinicians. We hope you enjoy reading both, and the article which describes a new assay for prohepcidin which proves to be decreased in haemochromatosis, supporting the idea that hepcidin is a key molecule in regulating iron absorption.
See page 735
While coeliac disease has usually been considered a T cell mediated disorder it is well recognised that coeliac have circulating antibodies to a widely distributed enzyme, transglutaminase 2 (TG2). Whether these antibodies exert important biological effects in vivo is uncertain but in vitro they certainly can. The present issue of Gut examined the binding of these antibodies to a wide range of tissues including normal jejunal biopsies from patients who subsequently developed coeliac disease
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