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Gut 2007;56:1646-1647; doi:10.1136/gut.2007.133926
Copyright © 2007 BMJ Publishing Group Ltd & British Society of Gastroenterology.

Commentaries

Gastric cancer

Secondary prevention of gastric cancer

Massimo Rugge

Correspondence to:
Massimo Rugge, Chair of Pathological Anatomy, Universitè degli Studi di Padova, Istituto Oncologico del Veneto IOV-IRCCS, Via Aristide Gabelli, 61, 35121, Padova, Italy; massimo.rugge@unipd.it


A matter of definitions


The first 150 words of the full text of this article appear below.

When I was young(er), my mentor, Pelayo Correa, who taught a generation of researchers orbiting around gastric oncology, encouraged me to spend some sabbatical time at the European Cancer Agency in Lyon "...to gain, among other things, a better perception of the crucial clinical impact that epidemiological data really have in clinical practice". I did not follow this good piece of advice... and it was a mistake! This was the first thought that came to me as I read the manuscript produced by de Vries et al in this issue of Gut (page 1665)—a well conducted study that will be mentioned by every paper addressing gastric precancerous lesions.1

The paper provides valuable insight on the declining prevalence of gastric precancerous lesions (that is, atrophic gastritis and gastric non-invasive neoplasia) in the Netherlands between 1991 and 2005: 15 years is a long enough time to give us . . . [Full text of this article]


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