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Neoplasia and cancer pathogenesis
* Cetuximab combined with multi-targeted sirna against HSP90, UBCH5, and SRC circumvented oncogene addiction, transactivation, and acquired resistance as a result of EGFR ubiquitination, and mutations/deletions in the kinase domain of EGFR in colorectal CA
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- * Cetuximab combined with multi-targeted sirna against HSP90, UBCH5, and SRC circumvented oncogene addiction, transactivation, and acquired resistance as a result of EGFR ubiquitination, and mutations/deletions in the kinase domain of EGFR in colorectal CA