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  • Fatty acid ethyl ester synthase inhibition ameliorates ethanol-induced Ca2+-dependent mitochondrial dysfunction and acute pancreatitis

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Pancreas
Original article
Fatty acid ethyl ester synthase inhibition ameliorates ethanol-induced Ca2+-dependent mitochondrial dysfunction and acute pancreatitis

Authors

  • Wei Huang Department of Cellular & Molecular Physiology, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK NIHR Liverpool Pancreas Biomedical Research Unit, RLBUHT, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK Department of Integrated Traditional Chinese and Western Medicine, Sichuan Provincial Pancreatitis Centre, West China Hospital, Sichuan University, China PubMed articlesGoogle scholar articles
  • David M Booth Department of Cellular & Molecular Physiology, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK PubMed articlesGoogle scholar articles
  • Matthew C Cane Department of Cellular & Molecular Physiology, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK PubMed articlesGoogle scholar articles
  • Michael Chvanov Department of Cellular & Molecular Physiology, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK NIHR Liverpool Pancreas Biomedical Research Unit, RLBUHT, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK PubMed articlesGoogle scholar articles
  • Muhammad A Javed Department of Cellular & Molecular Physiology, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK NIHR Liverpool Pancreas Biomedical Research Unit, RLBUHT, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK PubMed articlesGoogle scholar articles
  • Victoria L Elliott NIHR Liverpool Pancreas Biomedical Research Unit, RLBUHT, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK PubMed articlesGoogle scholar articles
  • Jane A Armstrong NIHR Liverpool Pancreas Biomedical Research Unit, RLBUHT, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK PubMed articlesGoogle scholar articles
  • Hayley Dingsdale Department of Cellular & Molecular Physiology, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK PubMed articlesGoogle scholar articles
  • Nicole Cash Department of Cellular & Molecular Physiology, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK PubMed articlesGoogle scholar articles
  • Yan Li NIHR Liverpool Pancreas Biomedical Research Unit, RLBUHT, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK PubMed articlesGoogle scholar articles
  • William Greenhalf NIHR Liverpool Pancreas Biomedical Research Unit, RLBUHT, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK PubMed articlesGoogle scholar articles
  • Rajarshi Mukherjee Department of Cellular & Molecular Physiology, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK NIHR Liverpool Pancreas Biomedical Research Unit, RLBUHT, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK PubMed articlesGoogle scholar articles
  • Bhupendra S Kaphalia Department of Pathology, University of Texas Medical Branch, Galveston, Texas, USA PubMed articlesGoogle scholar articles
  • Mohammed Jaffar Morvus Technology Limited, Carmarthen, UK PubMed articlesGoogle scholar articles
  • Ole H Petersen Cardiff School of Biosciences, University of Cardiff, Cardiff, UK PubMed articlesGoogle scholar articles
  • Alexei V Tepikin Department of Cellular & Molecular Physiology, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK PubMed articlesGoogle scholar articles
  • Robert Sutton NIHR Liverpool Pancreas Biomedical Research Unit, RLBUHT, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK PubMed articlesGoogle scholar articles
  • David N Criddle Department of Cellular & Molecular Physiology, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK NIHR Liverpool Pancreas Biomedical Research Unit, RLBUHT, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside, UK PubMed articlesGoogle scholar articles
  1. Correspondence to Dr David N Criddle, Department of Cellular & Molecular Physiology/NIHR Pancreas Biomedical Research Unit, RLBUHT, Institute of Translational Medicine, University of Liverpool, Liverpool, Merseyside L693BX, UK; criddle{at}liv.ac.uk
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Citation

Huang W, Booth DM, Cane MC, et al
Fatty acid ethyl ester synthase inhibition ameliorates ethanol-induced Ca2+-dependent mitochondrial dysfunction and acute pancreatitis
Gut 2014;63:1313-1324.

Publication history

  • Received November 1, 2012
  • Revised August 30, 2013
  • Accepted September 15, 2013
  • First published October 25, 2013.
Online issue publication 
October 26, 2017

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Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions This is an Open Access article distributed in accordance with the terms of the Creative Commons Attribution (CC BY 3.0) license, which permits others to distribute, remix, adapt and build upon this work, for commercial use, provided the original work is properly cited. See: http://creativecommons.org/licenses/by/3.0/