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  • Colesevelam attenuates cholestatic liver and bile duct injury in Mdr2−/− mice by modulating composition, signalling and excretion of faecal bile acids

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Hepatology
Original article
Colesevelam attenuates cholestatic liver and bile duct injury in Mdr2−/− mice by modulating composition, signalling and excretion of faecal bile acids

Authors

  • Claudia Daniela Fuchs Hans Popper Laboratory of Molecular Hepatology, Division of Gastroenterology and Hepatology, Department of Internal Medicine III, Medical University of Vienna, Vienna, Austria PubMed articlesGoogle scholar articles
  • Gustav Paumgartner Hans Popper Laboratory of Molecular Hepatology, Division of Gastroenterology and Hepatology, Department of Internal Medicine III, Medical University of Vienna, Vienna, Austria PubMed articlesGoogle scholar articles
  • Veronika Mlitz Hans Popper Laboratory of Molecular Hepatology, Division of Gastroenterology and Hepatology, Department of Internal Medicine III, Medical University of Vienna, Vienna, Austria PubMed articlesGoogle scholar articles
  • Victoria Kunczer Hans Popper Laboratory of Molecular Hepatology, Division of Gastroenterology and Hepatology, Department of Internal Medicine III, Medical University of Vienna, Vienna, Austria PubMed articlesGoogle scholar articles
  • Emina Halilbasic Hans Popper Laboratory of Molecular Hepatology, Division of Gastroenterology and Hepatology, Department of Internal Medicine III, Medical University of Vienna, Vienna, Austria PubMed articlesGoogle scholar articles
  • Nadja Leditznig Hans Popper Laboratory of Molecular Hepatology, Division of Gastroenterology and Hepatology, Department of Internal Medicine III, Medical University of Vienna, Vienna, Austria PubMed articlesGoogle scholar articles
  • Annika Wahlström Department of Molecular and Clinical Medicine, Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden PubMed articlesGoogle scholar articles
  • Marcus Ståhlman Department of Molecular and Clinical Medicine, Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden PubMed articlesGoogle scholar articles
  • Andrea Thüringer Institute of Pathology, Medical University of Graz, Graz, Austria PubMed articlesGoogle scholar articles
  • Karl Kashofer Institute of Pathology, Medical University of Graz, Graz, Austria PubMed articlesGoogle scholar articles
  • Tatjana Stojakovic Clinical Institute of Medical and Chemical Laboratory Diagnostics, Medical University of Graz, Graz, Austria PubMed articlesGoogle scholar articles
  • Hanns-Ulrich Marschall Department of Molecular and Clinical Medicine, Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden PubMed articlesGoogle scholar articles
  • Michael Trauner Hans Popper Laboratory of Molecular Hepatology, Division of Gastroenterology and Hepatology, Department of Internal Medicine III, Medical University of Vienna, Vienna, Austria PubMed articlesGoogle scholar articles
  1. Correspondence to Professor Michael Trauner, Division of Gastroenterology and Hepatology, Department of Internal Medicine III, Medical University of Vienna, Vienna 1090, Austria; michael.trauner{at}meduniwien.ac.at
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Citation

Fuchs CD, Paumgartner G, Mlitz V, et al
Colesevelam attenuates cholestatic liver and bile duct injury in Mdr2−/− mice by modulating composition, signalling and excretion of faecal bile acids
Gut 2018;67:1683-1691.

Publication history

  • Received May 24, 2017
  • Revised February 20, 2018
  • Accepted March 3, 2018
  • First published April 10, 2018.
Online issue publication 
November 24, 2018

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© Article author(s) (or their employer(s) unless otherwise stated in the text of the article) 2018. All rights reserved. No commercial use is permitted unless otherwise expressly granted. This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/