Regular ArticlePolymorphisms of the ICAM-1 Gene Are Associated with Inflammatory Bowel Disease, Regardless of the p-ANCA Status
References (30)
- et al.
Cell adhesion in the immune system
Immunol. Today
(1993) - et al.
Intercellular adhesion molecule-1 expression and cell signaling cascades
Free Rad. Biol. Med.
(2000) - et al.
Polymorphisms and linkage analysis for ICAM-1 and the selectin gene cluster
Genomics
(1994) - et al.
Adhesion molecule polymorphisms in chronic renal allograft failure
Kidney Int.
(1999) - et al.
Intercellular adhesion molecule 1 gene associations with immunologic subsets of inflammatory bowel disease
Gastroenterology
(1995) - et al.
Neutrophil autoantibodies in ulcerative colitis: Familial aggregation and genetic heterogeneity
Gastroenterology
(1992) - et al.
Distinct associations of HLA class II genes with inflammatory bowel disease
Gastroenterology
(1993) - et al.
A distinct subset of antineutrophil cytoplasmic antibodies is associated with inflammatory bowel disease
J. Allergy Clin. Immunol.
(1990) - et al.
Neutrophil autoantibodies: A genetic marker in primary sclerosing cholangitis and ulcerative colitis
Gastroenterology
(1994) - et al.
Nonradioactive HLA class II typing using polymerase chain reaction and digoxigenin-11-2′-3′-dideoxy-uridinetriphosphate-labeled oligonucleotide probes
Hum. Immunol.
(1991)
Antineutrophil antibodies in familial inflammatory bowel disease
Gastroenterology
Transendothelial migration
Inhibition of human mixed lymphocyte reactions by monoclonal antibodies to intercellular adhesion molecule-1 (ICAM-1)
Intercellular adhesion molecule-2, a second counter-receptor for CD11a/CD18 (leukocyte function-associated antigen-1), provides a costimulatory signal for T-cell receptor-initiated activation of human T cells
J. Immunol.
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2007, Journal of Surgical ResearchCitation Excerpt :The importance of ICAM-1 expression has been demonstrated by clinical trials of ICAM-1 antisense oligonucleotides that have shown promise in treating active CD [20, 21]. Crohn’s disease is associated with polymorphisms of the ICAM-1 gene, which might therefore represent a functional candidate gene [22]. The anti-inflammatory properties of VIP are exemplified by its ability to inhibit immune cells functions, cell migration, production of proinflammatory cytokines, chemokines, and inducible nitric oxide synthase (NOS) and to enhance the production of anti-inflammatory cytokines IL-10 and IL-1Ra [23, 24].
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