Increased risk of bacterial endocarditis in inflammatory bowel disease
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Cited by (40)
Presumptive complicating Clostridium paraputrificum bacteremia as a presenting manifestation in a patient with undiagnosed ulcerative colitis followed by acute colonic pseudo-obstruction
2023, IDCasesCitation Excerpt :In the setting of inflammatory bowel diseases, bacteremia has multiple etiologies. Defective integrity of the bowel mucosa and frequent endoscopic interventions in immunocompromised individuals are considered the primary causes [17]. In addition, an age of > 65 years and multiple comorbidities are associated with an increased risk of bacteremia in hospitalized patients with inflammatory bowel diseases [18].
Neurology of inflammatory bowel disease
2021, Journal of the Neurological SciencesCitation Excerpt :Ischemic events can be both cerebral and ocular, including ischemic optic neuropathy. Ischemic stroke occurs through several mechanisms: (1) large artery disease, including carotid occlusion due to atheroma, carotid thrombus [37] and dissection; (2) small vessel disease, e.g., corona radiata [38] or pontine lacunar infarcts; (3) cardioembolism, related to (a) paradoxical embolism though a patent foramen ovale in patients with lower limb, pelvic, mesenteric or vena cava venous thrombosis [39], (b) endocarditis [40,41], (c) atrial fibrillation; (4) vasculitis; (5) complication of anti-TNF-a therapy. Systemic and organ-specific vasculitis, namely cerebral, has been reported in association with IBD, especially with ulcerative colitis [42,43].
Endocarditis and Intravascular Infections
2014, Mandell, Douglas, and Bennett's Principles and Practice of Infectious DiseasesNeurologic manifestations of inflammatory bowel diseases
2014, Handbook of Clinical NeurologyCitation Excerpt :Ischemic events can be both cerebral and ocular. Ischemic stroke occurs through several mechanisms: (1) large artery disease, including even a case of common carotid occlusion; (2) small vessel disease, e.g., corona radiata (Ogawa et al., 2011) or pontine lacunar infarcts; (3) cardioembolism, related to (a) paradoxical embolism though a patent foramen ovale in patients with lower limb, pelvic, or mesenteric venous thrombosis, either symptomatic or not, (b) endocarditis (Kreuzpaintner et al., 1992); (4) vasculitis. A few cases of arterial ischemic complications have been reported as a complication of anti-TNF-α therapy (Vannucchi et al., 2011; Cohen et al., 2012).
Cardiobacterium hominis endocarditis presenting as acute embolic stroke: A case report and review of the literature
2011, Heart and Lung: Journal of Acute and Critical CareCitation Excerpt :His previous prednisolone therapy could also have been a risk factor for this infection. We could not rule out his Crohn's disease as a risk factor for C. hominis endocarditis, because inflammatory bowel disease is a known risk factor for infective endocarditis.29 Cardiobacterium hominis can also be found among gastrointestinal-tract flora, and a translocation of this bacterium from the gastrointestinal tract into the bloodstream could have occurred during unknown previous active inflammatory bowel disease in our patient.
Relapsing meningitis in a 69-year-old male
2011, Medicina Clinica