Biochemical and Biophysical Research Communications
Volume 184, Issue 2, 30 April 1992, Pages 680-685
Differential distribution of nitric oxide synthase between cell fractions isolated from the rat gastric mucosa
References (11)
- et al.
Pharmacol. Rev
(1991) - et al.
Biochem. J
(1990) - et al.
Gastroenterology
(1991) - et al.
Eur. J. Pharmacol
(1989) - et al.
Br. J. Pharmacol
(1990)
There are more references available in the full text version of this article.
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