Changes of lipid peroxide levels in blood and liver tissue of patients with obstructive jaundice
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2016, Asian Pacific Journal of Tropical MedicineCitation Excerpt :The pathogenesis of cholestatic liver injury caused by bile duct obstruction or functional defect in bile formation is thought to have a critical role in extra-hepatic damage [1]. Stagnant bile acid causes hepatotoxicity, disrupts hepatocyte cell membranes, and releases reactive oxygen species from peritoneal neutrophil in cholestatic liver diseases with lipid peroxidation [2,3]. Intra-hepatic accumulation of bile acid and toxic materials causes pro-inflammation and oxidative stress, leading to fibrosis and cirrhosis [4].
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Effects of ganoderma lucidum on obstructive jaundice-induced oxidative stress
2010, Asian Journal of SurgeryPlasma superoxide radical in jaundiced patients and role of xanthine oxidase
2008, American Journal of the Medical SciencesCitation Excerpt :Despite the clear importance of the results obtained from experimental studies under this perspective, there are very limited clinical investigations on this issue. To the best of our knowledge, there are only 3 related clinical studies in the literature; the first showed that patients with obstructive jaundice present high lipid peroxide levels in their blood and liver,22 the second showed that cholestasis, besides concurring with increased hepatic lipid peroxidation, is associated with a decreased protein and nonprotein sulfhydryl content in the liver. These oxidative parameters are reversed almost completely after biliary drainage.23
Ghrelin alleviates biliary obstruction-induced chronic hepatic injury in rats
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