ORIGINAL ARTICLESEffect of screening for hepatitis C virus antibody and hepatitis B virus core antibody on incidence of post-transfusion hepatitis
References (12)
- et al.
Measurement of anti-GOR antibodies in prevention of post-transfusion non-A, non-B hepatitis
Lancet
(1991) - et al.
Post-transfusion hepatitis in Australia: report of the Australian Red Cross Study
Lancet
(1982) Report of Post-Transfusion Hepatitis Research Group, Tokyo
(1987)- H. Iizuka, K. Ohmura, A. Ishijima, et al., Correlation between anti-HBc titres and HBV DNA in blood units without...
- et al.
Post-transfusion fulminant hepatitis B associated with precore-defective HBV mutants
Vox Sang
(1991)
Cited by (203)
Multicenter evaluation of the new Alinity s anti-HCV II assay for routine hepatitis C virus blood screening
2023, Journal of Clinical Virology PlusHigh Frequency of Cardiovascular Risk Factors in Overweight Adult Japanese Subjects
2007, Archives of Medical ResearchCitation Excerpt :Therefore, we defined NASH as ALT ≥35 IU/l, ALT/AST ≥1.0 and alcohol consumption <20 g/day (18) in the present study, to exclude alcoholic liver disease. As we had no patient data on other forms of chronic liver disease, our definition of NASH may have included a small number of subjects infected with viral hepatitis B or C, estimated at 2% prevalence in Japanese adults (23–24). Our results indicate that “obesity” and “overweight” conditions in Japanese are associated with an increased prevalence of a wide range of obesity-related diseases including hypertension, dyslipidemia, glucose intolerance, hyperuricemia, NASH and inflammation.
Occult hepatitis B virus infection
2007, Journal of HepatologyCitation Excerpt :Thus, the occult infection appears to be mostly due to a strong suppression of viral replication and gene expression affecting viruses whose genetic variability is comparable to that of HBV strains from individuals with “overt” chronic HBV infection [23]. Relevant though indirect confirmation on this assumption is provided by the observations that occult HBV may be transmitted (experimentally to chimpanzees and by blood transfusion or organ transplantation to humans) inducing classic acute hepatitis B in the recipients [24–29] and that occult HBV carriers may show an acute reactivation of the infection with the reappearance of the typical hepatitis B serological profile [30–34]. The mechanisms responsible for the inhibition of HBV activities remain at present largely obscure, and all the hypotheses proposed are essentially based on indirect evidence.
Timing of interferon therapy and sources of infection in patients with acute hepatitis C
2006, Hepatology ResearchRisk factors for hepatitis B and C in multi-transfused patients in Uruguay
2005, Journal of Clinical Virology
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STUDY PARTICIPANTS: K. Okochi, MD, S. Inaba, MD, K. Tokunaga, BSc (Fukuoka), H. Yoshizawa, MD, H. Moori, MD, M. Mizui, BSc (Hiroshima); S. Ito, MD, F. Yokoishi, BSc (Tokushima); K. Tsubaki, MD, H. Yamano, BSc (Osaka), K. Konishi, MD, K. Kanemitsu, BSc (Kanazawa); M. Shimizu, MD, J. Watanabe, BSc, N Nojiri, BSc, K, Nishioka, MD (Tokyo); T. Tanaka, BSc (Saitama); S. Kikuchi, MD, N. Endo, BSc (Sendai). Correspondence to Dr K. Nishioka, Japanese Red Cross Central Blood Centre, 4-1-31, Hiroo, Shibuya-Ku, Tokyo 150, Japan.