ORIGINAL ARTICLESEffect of Helicobacter pylori on gastric somatostatin in duodenal ulcer disease
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Mechanisms of Helicobacter pylori-Induced Gastric Inflammation
2018, Physiology of the Gastrointestinal Tract, Sixth EditionRedox signaling in the gastrointestinal tract
2017, Free Radical Biology and MedicineCitation Excerpt :This is due to H. pylori urease activity that produces ammonia from urea, which neutralizes HCl on the gastric mucosa. As a result, an inadequate amount of somatostatin is released increasing gastrin release and acid production by gastric mucosa [159,160]. Furthermore, H. pylori releases chemotactic factors, such as N-formyl-methionyl-leucyl phenylalanine peptide, and causes an inflammatory response in gastric mucosa, promoting IL-8 and IL-1β release from gastric epithelial cells [161,162].
Helicobacter pylori and Other Gastric Helicobacter Species
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2017, The LancetMucosal distribution of somatostatin-secreting gastric Delta cells in children with gastrointestinal reflux diseases
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