Intracisternal injection of peptide YY inhibits gastric emptying in rats
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Cited by (15)
Peptide YY
2016, The Curated Reference Collection in Neuroscience and Biobehavioral PsychologyPeripheral administration of PYY<inf>3-36</inf> produces conditioned taste aversion in mice
2005, Cell MetabolismCitation Excerpt :This may reflect that even though an intact vagus nerve is not required for the short-term anorexigenic actions of peripheral PYY3–36, unlike CCK and ghrelin, intact vagal tone could modulate the duration of action of PYY3–36. Moreover, it has been shown in the literature that intact efferent vagal parasympathetic tone modulates basal and food-induced release of PYY (Zhang et al., 1993), as well as some of the ability of the peptide to modulate gastrointestinal (GI) transit time and glandular secretion (Chen et al., 1996; Masuda et al., 1994). In contrast to the results reported here, a recent study has demonstrated blockade of PYY 3–36-mediated inhibition of food intake in the rat by bilateral subdiaphragmatic vagotomy (BSDV) (Koda et al., 2005).
Central and peripheral regulation of gastric acid secretion by peptide YY
2002, PeptidesCitation Excerpt :The observations that PYY is a potent inhibitor on cephalic phase acid secretion [36,65] and the inhibition is vagally dependent [50] suggest that the inhibitory effect of PYY on gastric acid secretion is neurally mediated, especially involving the vagus nerve. Recent studies revealed that PYY acts in the medullary dorsal vagal complex (DVC) to modulate gastric functions [11,12,13,14,90,92,95]. Both inhibitory and excitatory actions of PYY were observed.
Chapter IX Neuropeptide Y, peptide YY and pancreatic polypeptide receptor proteins and mRNAs in mammalian brains
2000, Handbook of Chemical Neuroanatomy