The control of homologous lysis

doi:10.1016/0167-5699(91)90005-E

Immunology Today

Volume 12, Issue 9, September 1991, Pages 312-315

Immunology Today

https://doi.org/10.1016/0167-5699(91)90005-E Get rights and content

Abstract

Complement activation unleashes powerful effector mechanisms against which host cells are protected by homologous restriction factors. These factors are glycolipid-anchored membrane proteins that either induce C3 convertase dissociation (for example decay-accelerating factor) or prevent the full development of the membrane attack complex (for example homologous restriction factor and CD59). In this article Peter Lachmann explores the biology and biochemistry o f these important and intriguing molecules.

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Mechanism Molecular mechanismThe control of homologous lysis

Abstract

Biochem. Biophys. Acta

Blood

Biochem. Biophys. Res. Commun.

J. Biol. Chem.

Cell. Immunol.

Mol. Immunol.

Biochem. Biophys. Res. Commun.

Blood

Immunol. Today

J. Exp. Med.

J. Immunol.

J. Exp. Med.

J. Exp. Med.

J. Exp. Med.

J. Exp. Med.

Immunology

Immunobiology

Mol. Immunol.

New Engl. J. Med.

Br. J. Haematol.

J. Immunol.

Mechanism Molecular mechanism
The control of homologous lysis