Research ReportPortal hemodynamics in chronic portal-systemic encephalopathy: Angiographic study in seven cases
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Cited by (39)
Portal Hypertension and Bleeding Esophageal Varices
2006, Zakim and Boyer's HepatologySelective embolization of the splenic vein in patients with hepatic encephalopathy and splenorenal shunt
2004, Journal of Vascular and Interventional RadiologyPredictors and implications of severe hypersplenism in patients with cirrhosis
2003, American Journal of the Medical SciencesCitation Excerpt :There does not seem to be significant correlation between portal pressure and the degree of hepatic decompensation. 7 The portal system has numerous collaterals that interconnect with systemic circulation, and the severity of portal hypertension depends upon size and number of portal-systemic collaterals irrespective of the liver function. 7 Furthermore, previous reports failed to establish a significant correlation between portal pressure and spleen size.8,9
Hepatic encephalopathy
2001, American Journal of GastroenterologyCitation Excerpt :The presence of large spontaneous portal-systemic shunts should be sought in selected patients with recurrent episodes of encephalopathy despite medical therapy, where a precipitating factor is not found. Large splenorenal or gastrorenal communications have been associated with episodes of “spontaneous” encephalopathy (absence of a precipitating factor) (58). Visualization of the collaterals can be obtained with ultrasound techniques and confirmed with visceral angiography.
Relationship between pre-TIPS liver perfusion by the portal vein and the incidence of post-TIPS chronic hepatic encephalopathy
2001, American Journal of GastroenterologyCitation Excerpt :Unlike the acute TIPS-induced hemodynamic changes, the natural history of liver cirrhosis is characterized by a progressive deterioration of liver function and by a slow increase in spontaneous portosystemic shunting. Chronic encephalopathy is rare in cirrhotic patients who were not treated by surgical shunts or TIPS (18–20). In these patients, large spontaneous portosystemic anastomoses are frequently observed (21).
Diagnosis and treatment of hepatic encephalopathy
2000, Best Practice and Research: Clinical Gastroenterology