Nitric oxide as a mediator of hemodynamic abnormalities and sodium and water retention in cirrhosis

N Engl J Med. 1998 Aug 20;339(8):533-41. doi: 10.1056/NEJM199808203390807.
No abstract available

Publication types

  • Review

MeSH terms

  • Animals
  • Ascites / etiology
  • Ascites / physiopathology
  • Extracellular Space / metabolism
  • Hemodynamics
  • Homeostasis
  • Humans
  • Hypertension, Portal / physiopathology
  • Kidney Diseases / physiopathology
  • Liver Cirrhosis / physiopathology*
  • Liver Cirrhosis, Experimental / physiopathology*
  • Nitric Oxide / antagonists & inhibitors
  • Nitric Oxide / physiology*
  • Nitric Oxide Synthase / antagonists & inhibitors
  • Nitric Oxide Synthase / metabolism
  • Sodium / metabolism*
  • Splanchnic Circulation / physiology

Substances

  • Nitric Oxide
  • Sodium
  • Nitric Oxide Synthase