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Limitations of non-ceruloplasmin-bound copper in routine clinical practice
  1. P J Twomey1,
  2. A Viljoen2,
  3. I M House3,
  4. T M Reynolds4,
  5. A S Wierzbicki5
  1. 1Department of Clinical Biochemistry, The Ipswich Hospital, Ipswich, UK
  2. 2Department of Chemical Pathology, Addenbrooke’s Hospital, Cambridge, UK
  3. 3The Medical Toxicology Unit Laboratory, Guy’s & St Thomas’ Hospital Trust, London, UK
  4. 4Department of Chemical Pathology, Queen’s Hospital, Burton-on-Trent, UK
  5. 5Department of Chemical Pathology, St Thomas’ Hospital, London, UK
  1. Correspondence to:
    P J Twomey
    Department of Clinical Biochemistry, The Ipswich Hospital, Heath Road, Ipswich IP4 5PD, UK;patrick.twomey{at}

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We read with interest the paper on Wilson’s disease by Merle et al (this issue, p 115). Some aspects of the investigation profile are omitted from the paper. Although data on the prevalence of reduced ceruloplasmin levels and raised non-ceruloplasmin-bound copper levels are provided outside cut-offs, data on mean (and median) levels, SD (interquartile ranges) and any skew or …

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  • Competing interests: None.

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