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Original article
Duodenal nutrient exclusion improves metabolic syndrome and stimulates villus hyperplasia

Authors

  • Kirk M Habegger Division of Endocrinology, Department of Internal Medicine, Metabolic Diseases Institute, Obesity Research Center, University of Cincinnati, Cincinnati, Ohio, USA PubMed articlesGoogle scholar articles
  • Omar Al-Massadi Division of Endocrinology, Department of Internal Medicine, Metabolic Diseases Institute, Obesity Research Center, University of Cincinnati, Cincinnati, Ohio, USA PubMed articlesGoogle scholar articles
  • Kristy M Heppner Division of Endocrinology, Department of Internal Medicine, Metabolic Diseases Institute, Obesity Research Center, University of Cincinnati, Cincinnati, Ohio, USA PubMed articlesGoogle scholar articles
  • Andriy Myronovych Division of Gastroenterology, Hepatology, and Nutrition, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, USA PubMed articlesGoogle scholar articles
  • Jenna Holland Division of Endocrinology, Department of Internal Medicine, Metabolic Diseases Institute, Obesity Research Center, University of Cincinnati, Cincinnati, Ohio, USA PubMed articlesGoogle scholar articles
  • Jose Berger Division of Endocrinology, Department of Internal Medicine, Metabolic Diseases Institute, Obesity Research Center, University of Cincinnati, Cincinnati, Ohio, USA PubMed articlesGoogle scholar articles
  • Chun-Xia Yi Institute for Diabetes and Obesity, Helmholtz Zentrum München and Technische Universität München, Munich, Germany PubMed articlesGoogle scholar articles
  • Yuanging Gao Division of Endocrinology, Department of Internal Medicine, Metabolic Diseases Institute, Obesity Research Center, University of Cincinnati, Cincinnati, Ohio, USA PubMed articlesGoogle scholar articles
  • Maarit Lehti Division of Endocrinology, Department of Internal Medicine, Metabolic Diseases Institute, Obesity Research Center, University of Cincinnati, Cincinnati, Ohio, USA PubMed articlesGoogle scholar articles
  • Nickki Ottaway Division of Endocrinology, Department of Internal Medicine, Metabolic Diseases Institute, Obesity Research Center, University of Cincinnati, Cincinnati, Ohio, USA PubMed articlesGoogle scholar articles
  • Sarah Amburgy Division of Endocrinology, Department of Internal Medicine, Metabolic Diseases Institute, Obesity Research Center, University of Cincinnati, Cincinnati, Ohio, USA PubMed articlesGoogle scholar articles
  • Christine Raver Division of Endocrinology, Department of Internal Medicine, Metabolic Diseases Institute, Obesity Research Center, University of Cincinnati, Cincinnati, Ohio, USA PubMed articlesGoogle scholar articles
  • Timo D Müller Institute for Diabetes and Obesity, Helmholtz Zentrum München and Technische Universität München, Munich, Germany PubMed articlesGoogle scholar articles
  • Paul T Pfluger Institute for Diabetes and Obesity, Helmholtz Zentrum München and Technische Universität München, Munich, Germany PubMed articlesGoogle scholar articles
  • Rohit Kohli Division of Gastroenterology, Hepatology, and Nutrition, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, USA PubMed articlesGoogle scholar articles
  • Diego Perez-Tilve Division of Endocrinology, Department of Internal Medicine, Metabolic Diseases Institute, Obesity Research Center, University of Cincinnati, Cincinnati, Ohio, USA PubMed articlesGoogle scholar articles
  • Randy J Seeley Division of Endocrinology, Department of Internal Medicine, Metabolic Diseases Institute, Obesity Research Center, University of Cincinnati, Cincinnati, Ohio, USA PubMed articlesGoogle scholar articles
  • Matthias H Tschöp Institute for Diabetes and Obesity, Helmholtz Zentrum München and Technische Universität München, Munich, Germany PubMed articlesGoogle scholar articles
  1. Correspondence to Professor Matthias Tschöp, Institute for Diabetes and Obesity, Helmholtz Zentrum München, German Research Center for Environmental Health (GmbH), Ingolstädter Landstr. 1, Neuherberg/Munich 85764, Germany; tschoep{at}helmholtz-muenchen.de
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Citation

Habegger KM, Al-Massadi O, Heppner KM, et al
Duodenal nutrient exclusion improves metabolic syndrome and stimulates villus hyperplasia

Publication history

  • Received January 28, 2013
  • Revised August 19, 2013
  • Accepted September 4, 2013
  • First published October 9, 2013.
Online issue publication 
July 08, 2014

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