PWE-114 Modulation of non-canonical nfkb pathways may underlie the anti-inflammatory, resolution-like activation of circulating cd14hi monocytes in hyper-acute liver failure

RD Abeles; CG Antoniades; DL Shawcross; W Khamri; GK Manakkat-Vijay; W Bernal; JA Wendon; D Vergani

doi:10.1136/gutjnl-2015-309861.563

Article info

Liver – Clinical Science

PWE-114 Modulation of non-canonical nfkb pathways may underlie the anti-inflammatory, resolution-like activation of circulating cd14hi monocytes in hyper-acute liver failure

Authors

RD Abeles 1Institute of Liver Studies, King–s College London at King–s College Hospital PubMed articles Google scholar articles
CG Antoniades 2Section of Hepatology, St Mary–s Hospital, Imperial College London, London, UK PubMed articles Google scholar articles
DL Shawcross 1Institute of Liver Studies, King–s College London at King–s College Hospital PubMed articles Google scholar articles
W Khamri 2Section of Hepatology, St Mary–s Hospital, Imperial College London, London, UK PubMed articles Google scholar articles
GK Manakkat-Vijay 1Institute of Liver Studies, King–s College London at King–s College Hospital PubMed articles Google scholar articles
W Bernal 1Institute of Liver Studies, King–s College London at King–s College Hospital PubMed articles Google scholar articles
JA Wendon 1Institute of Liver Studies, King–s College London at King–s College Hospital PubMed articles Google scholar articles
D Vergani 1Institute of Liver Studies, King–s College London at King–s College Hospital PubMed articles Google scholar articles

Citation

Abeles R, Antoniades C, Shawcross D, et al

PWE-114 Modulation of non-canonical nfkb pathways may underlie the anti-inflammatory, resolution-like activation of circulating cd14hi monocytes in hyper-acute liver failure

Gut 2015;64:A262-A263.

Publication history

First published June 22, 2015.

Online issue publication

April 18, 2016

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