No patients and controls | |||||
Origin of E coli strain | |||||
n | ER 2-a | CIL 2-b | CDH 2-c | Controls2-d | |
A1 | 26 | 12e | 4 | 2 | 8 |
A2 | 11 | 2e | 7 | 1 | 1 |
B | 4 | — | 1 | — | 3 |
C | 5 | 1 | 1 | 1 | 2 |
D | 1 | 1 | — | — | — |
E | 1 | — | — | 1 | — |
F | 3 | — | 2 | 1 | — |
G | 1 | 1 | — | — | — |
H | 3 | — | — | — | 3 |
I | 5 | 3 | — | 1 | 1 |
J | 1 | 1 | — | — | — |
K | 1 | 1 | — | — | — |
L | 4 | 3 | — | 1 | — |
M | 1 | — | — | — | 1 |
N | 1 | — | — | — | 1 |
O | 1 | — | — | — | 1 |
No patients or controlse | 61f | 20g | 14g | 7 | 21 |
↵2-a ER, early recurrent ileal lesion;
↵2-b CIL, chronic ileal lesion;
↵2-c CDH, healthy ileal mucosa from CD patients without recurrent lesion;
↵2-d controls, biopsy specimens from patients with non-inflammatory bowel disease and stools from healthy non-hospitalised individuals;
2-e one patient was considered twice because he presented early endoscopic recurrence at two and three years after surgery;
2-f some patients harboured more than oneE coli isolate distributed in several groups;
2-g another patient was also considered twice because he was operated on for CD (chronic lesion) and presented early endoscopic recurrence after surgery.