Spontaneous, heritable colitis in a new substrain of C3H/HeJ mice
References (22)
- et al.
Colonic adenocarcinoma associated with chronic colitis in cotton top marmosets, Saguinus oedipus
Gastroenterology
(1981) - et al.
Characterization of spontaneous colitis in cotton-top tamarins (Saguinus oedipus) and its response to sulfasalazine
Gastroenterology
(1985) - et al.
Alopecia areata in aging C3H/HeJ mice
J Invest Dermatol
(1994) - et al.
Ulcerative colitis-like disease in mice with a disrupted interleukin-2 gene
Cell
(1993) - et al.
Interleukin-10-deficient mice develop chronic enterocolitis
Cell
(1993) - et al.
Experimental colitis in animal models
Scand J Gastroenterol
(1992) Conceptual evaluation of inbred laboratory mouse models as tools for the study of diseases in other species
Lab Anim
(1992)- et al.
The “Swiss roll”: a simple technique for histologic studies of the rodent intestine
Lab Anim
(1981) - et al.
Catalog of mutant genes and polymorphic loci
Cited by (184)
Quantitative Trait Locus and Integrative Genomics Revealed Candidate Modifier Genes for Ectopic Mineralization in Mouse Models of Pseudoxanthoma Elasticum
2019, Journal of Investigative DermatologyAlopecia areata: A multifactorial autoimmune condition
2019, Journal of AutoimmunityGnotobiotics and Inflammatory Bowel Disease
2017, GnotobioticsUncovering Pathogenic Mechanisms of Inflammatory Bowel Disease Using Mouse Models of Crohn's Disease–Like Ileitis: What is the Right Model?
2017, Cellular and Molecular Gastroenterology and HepatologyExperimental colitis models: Insights into the pathogenesis of inflammatory bowel disease and translational issues
2015, European Journal of PharmacologyAnimal models of gastrointestinal inflammation and cancer
2014, Life SciencesCitation Excerpt :Colitis spontaneously occurs in these mouse strains. C3H/HeJBir, which was selectively bred from C3H/HeJ with colitis symptoms, has higher B cell and T cell reactivity to enteric bacterial antigens (Elson et al., 2000; Sundberg et al., 1994). Genetic analysis showed that the high incidence of colitis involves multiple genes and is inherited as a quantitative trait (Mahler et al., 1998).
View full text
Copyright © 1994 Published by Elsevier Inc.