Survey of human and rat microsatellites
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2018, Advances in Quantum ChemistryCitation Excerpt :Based on availability of “high resolution,” enzyme quantum reader23 measurements, δt ≪ 10− 13 s, of intramolecular entangled proton qubit states,36–39 |+ 〉 ⇄ |−〉, the ambient temperature, in vivo antientanglement hypothesis4,5 is falsified. Since EPR-generated entangled proton qubits occupy decoherence-free subspaces40–42 between two indistinguishable sets of electron lone pairs (Figs. 1–3), and enzyme–proton entanglement quantum processing reactions satisfy Δt′ ≤ 10− 14 s,36–39,87 ion incursions, H2O, and random temperature fluctuations101 do not obstruct evolutionarily selected enzyme–proton entanglement quantum processing measurements19–22; otherwise, the EPR-entanglement algorithm36,37 could not accurately predict the relative evolutionary distributions of the 22 most abundant microsatellites common to “evolving” rat and human genomes,46 which it does very well.36,39 Additionally, the quantum entanglement algorithm provides entanglement-enabled molecular dynamic explanations for expansions and contractions exhibited by Huntington's disease (CAG)n-repeat evolution,22,52 and further, provides testable explanations for the ~ 2 to ~ 12 years delay in phenotypic manifestation of Huntington's disease,38 after inheriting a “long” (CAG)70 repeat within the huntingtin gene.
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