Upper intestinal surveillance in familial adenomatous polyposis
References (36)
- et al.
Upper gastrointestinal cancer in patients with familial adenomatous polyposis
Lancet
(1989) - et al.
Natural history of fundic gland polyposis in patients with familial adenomatosis coli/Gardner's syndrome
Gastroenterology
(1985) - et al.
Upper gastrointestinal cancer in familial adenomatous polyposis
Lancet
(1988) - et al.
The risk of upper gastrointestinal cancer in familial adenomatous polyposis
Gastroenterology
(1992) - et al.
Restriction of ocular fundus lesions to a specific subgroup of APC mutations in adenomatous polyposis coli patients
Cell
(1993) - et al.
Phenotypic variability of familial adenomatous polyposis in 11 untreated families with identical APC gene mutation
Gastroenterology
(1994) - et al.
Natural history of duodenal lesions in Japanese patients with familial adenomatosis coli (Gardner's syndrome)
Gastroenterology
(1989) - et al.
Upper gastrointestinal polyps in Gardner's syndrome
Gastroenterology
(1984) - et al.
Alleles of the APC gene: an attenuated form of familial polyposis
Cell
(1993) - et al.
Staging of pancreatic and ampullary carcinoma by endoscopic ultrasonography. Comparison with conventional sonography, computed tomography, and angiography
Gastroenterology
(1992)
Gastroduodenal polyps in patients with familial adenomatous polyposis
Dis Colon Rectum
Gastroduodenal polyps in familial polyposis coli
Dis Colon Rectum
Gastroduodenal polyps in familial adenomatous and juvenile polyposis
Endoscopy
Gastric and duodenal polyps in familial adenomatous polyposis: a prospective study of the nature and prevalence of upper gastrointestinal polyps
Gut
Risk of gastric cancer among Korean familial adenomatous polyposis (FAP) patients (meeting abstract)
The impact of familial adenomatous polyposis on the tumorigenesis and mortality at the several organs: its rational treatment
Ann Surg
Gastric lesions of familial polyposis coli
Cancer
The upper gastrointestinal tract
Cited by (65)
Hereditary cancer syndromes as model systems for chemopreventive agent development
2016, Seminars in OncologyCitation Excerpt :Progression to colorectal cancer occurs with nearly complete penetrance by age 40–50 years; however, malignancy in childhood does occur [65]. Patients with FAP may also develop upper gastrointestinal polyps, although these less commonly progress to malignancy as compared to colon and rectal adenomas [66–68]. Sites other than the gastrointestinal tract are at increased risk of tumors and malignancy including: thyroid, liver, brain, bone, adrenal gland, retinal pigment, and subcutaneous tissues.
The natural history of familial adenomatous polyposis syndrome: A 24 year review of a single center experience in screening, diagnosis, and outcomes
2014, Journal of Pediatric SurgeryCitation Excerpt :In the modern era, most FAP patients are not developing colorectal cancer due to the widespread practice of early colectomy. Therefore, secondary causes of morbidity, such as duodenal adenocarcinoma are being encountered in the post-colectomy population [5]. More than half of our patients (59%) developed upper gastrointestinal (UGI) polyposis, with mean onset at age 17 years.
Endoscopic Management of Upper Gastrointestinal Familial Adenomatous Polyposis Syndrome and Ampullary Tumors
2011, Clinical Gastrointestinal Endoscopy, Second EditionColonic Polyps and Polyposis Syndromes
2010, Sleisenger and Fordtran’s Gastrointestinal and Liver Disease- 2 Volume Set: Pathophysiology, Diagnosis, Management, Expert Consult Premium Edition - Enhanced Online Features and PrintColorectal Cancer: Pathogenesis and Risk Factors
2009, CT Colonography: Principles and Practice of Virtual ColonoscopyGastrointestinal oncology. What should be the monitoring strategy for duodenal adenomas in familial adenomatous polyposis?
2008, Gastroenterologia y Hepatologia