Gastroenterology

Gastroenterology

Volume 144, Issue 2, February 2013, Pages 265-269
Gastroenterology

Editorial
Preventing Pancreatitis by Protecting the Mitochondrial Permeability Transition Pore

https://doi.org/10.1053/j.gastro.2012.12.010Get rights and content

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Acknowledgments

The authors wish to thank Andreas Holtz for secretarial assistance.

References (33)

  • O. Sadr-Azodi et al.

    Cigarette smoking, smoking cessation and acute pancreatitis: a prospective population-based study

    Gut

    (2012)
  • D.C. Whitcomb et al.

    Common genetic variants in the CLDN2 and PRSS1-PRSS2 loci alter risk for alcohol-related and sporadic pancreatitis

    Nat Genet

    (2012)
  • M.M. Lerch et al.

    Models of acute and chronic pancreatitis

    Gastroenterology

    (2013)
  • N. Shalbueva et al.

    Effects of oxidative alcohol metabolism on mitochondrial permeability transition pore and necrosis in a mouse model of alcoholic pancreatitis

    Gastroenterology

    (2012)
  • M. Lampel et al.

    Acute interstitial pancreatitis in the rat induced by excessive doses of pancreatic secretagogue

    Virchows Arch A Path Anat Hist

    (1977)
  • T. Hirano et al.

    Apical secretion of lysosomal enzymes in rabbit pancreas occurs via a secretagogue regulated pathway and is increased after pancreatic duct obstruction

    J Clin Invest

    (1991)
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    Conflicts of interest The authors disclose no conflicts.

    Funding The authors' original work is supported by the Alfried-Krupp-von-Bohlen-und-Hahlbach-Foundation (Graduate Schools Tumour Biology and Free Radical Biology), the Deutsche Krebshilfe/Dr Mildred-Scheel-Stiftung (109102), the Deutsche Forschungsgemeinschaft (DFG GRK840-E3/E4, MA 4115/1-2/3, NI 1297/1-1), the Federal Ministry of Education and Research (BMBF GANI-MED 03152061A and BMBF 0314107), and the European Union (EU-FP-7: EPC-TM and EU-FP7-REGPOT-2010-1).

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