Visceral hypersensitivity is currently considered a key pathophysiological mechanism
involved in pain perception in large subgroups of patients with functional gastrointestinal …

The potential relevance of systemic and gastrointestinal immune activation in the
pathophysiology and symptom generation in the irritable bowel syndrome (IBS) is supported …

Background & Aims Mediators released by the intestinal mucosa of patients with irritable
bowel syndrome (IBS) affect the function of enteric and extrinsic sensory nerves, which can …

There is increasingly convincing evidence supporting the participation of the gut
microenvironment in the pathophysiology of irritable bowel syndrome (IBS). Studies …

Background Evidence suggests a role of intestinal microbiota-host interactions in the
pathophysiology and symptoms of irritable bowel syndrome (IBS). Objective The objective of …

Background Intestinal immune activation is involved in irritable bowel syndrome (IBS)
pathophysiology. While most dietary approaches in IBS involve food avoidance, there are …

Background & Aims Acute infectious gastroenteritis increases the risk for irritable bowel
syndrome (IBS) and functional dyspepsia (FD). Children are particularly vulnerable to …

Objective Non-coeliac gluten sensitivity (NCGS) is characterised by intestinal and
extraintestinal symptoms related to the ingestion of gluten-containing foods, in the absence …

Irritable bowel syndrome (IBS), a common functional gastrointestinal disorder, is classified
according to bowel habits as IBS with constipation (IBS‐C), with diarrhea (IBS‐D), with …

It has been found that the presence of a high number of leukocytes and the presence of lung
alterations may identify those patients with severe acute pancreatitis [5]; in fact, using a cut …